I too wonder if it is due to the way it was consumed primarily (smoking).
There is a bit about the impact of cannabinoids on the cardiovascular system however.
> The impact of exogenous cannabinoids on the cardiovascular system has been well described.7,33 After acute exposure, cannabis induces dose-dependent tachycardia and, in some cases, decreased ventricular contractility, palpitations, atrial fibrillation and arrhythmia.
Table 2 differentiates the form of consumption but I couldn't find them controlling for it. Did I miss that somehow?
In the "Interpretation" section of the paper they specify: "Although a similarly elevated
odds of history of heart attacks was observed across methods of recent cannabis consumption, only smoking as a primary method achieved
statistical significance."
>After acute exposure, cannabis induces dose-dependent tachycardia and, in some cases, decreased ventricular contractility, palpitations, atrial fibrillation and arrhythmia
i feel like this is saying what a lot of us already know about cannabis usage, more specifically about THC as this won't be found as much in low THC strains: higher doses have a tendency to cause anxiety sometimes. all of those symptoms tend to be, at least in my experience, pretty comorbid with a state of high anxiety, and more often than not, I would think, the more easily observable ones than the neurological or psychological effects associated therewith.
There is a bit about the impact of cannabinoids on the cardiovascular system however.
> The impact of exogenous cannabinoids on the cardiovascular system has been well described.7,33 After acute exposure, cannabis induces dose-dependent tachycardia and, in some cases, decreased ventricular contractility, palpitations, atrial fibrillation and arrhythmia.
Table 2 differentiates the form of consumption but I couldn't find them controlling for it. Did I miss that somehow?