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While this is always something to consider, generally, 6-fold increase in risk due to exposure is likely not just a population demographic artifact.

TCE and its relation to Parkinson's has been studied for at least the last 15 years. Though I haven't come across anything definitive on the mechanism of action, there seems to be indication that a combination of TCE or its analogues combined with other risk factors (assumed to be genetic) generates substantial progressive dopaminergic neuron loss, a hallmark of Parkinson's disease progression.



> 6-fold increase in risk due to exposure

Interestingly, the Guardian article does not cite a source for that claim, so it is impossible to evaluate it soundly.

However, just above, it states:

>> the number of people with Parkinson’s has increased 35% the last 10 years

which is completely in line with the increase in at risk population + improved probability of diagnosis.

That doesn't mean there is no link.




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