Given mass distribution of our galaxy is decidedly anisotropic I would expect the answer to be a resounding "no". The ((5/57)/ (4 Pi)) implicity assumes uniform incoming trajectories - I think. There's an odd mixing of radians and steradians in this formula, if the "4 Pi" represents the total solid angle of a sphere.
I suggest you read the paper. It's packed with subjective language (Row one of table one, the incoming trajectory is said to be "virtually" in the ecliptic plane), and has so many unstated assumptions behind their so-called significance of the trajectory it's not valid.
And the stated 5 degree angle disagrees with published observations.
And the estimate of the size is over double the confirmed observations of size.
While "Not aligning with scientific consensus doesn't make your suggestions worthless" is true, ignoring published results does.
His argument regarding the trajectory into our Solar System is pretty flaky. It completely disregards Hopkin's computation of a "steep entry angle" and supposition that it comes from the "thick disk", instead assuming the incoming trajectories of interstellar objects are uniformly distributed across the celestial sphere.
Mass distribution in our galaxy is decidedly anisotropic - most mass lies in the galatic plane.
Loeb's estimate of the comet size is strange, when two observatories concur that the maximum size is around 10km.
Yep, keep how to make PCP in a bathtub away from kids. Keep holocaust deniers away from kids. Keep race-hate videos away from kids.
Keep Hancock and Joe Rogan away from kids.
Keep lies and conspiracy theories away from kids.
You seem to think kids are good at risk analysis and critical thinking. There are exceptions, but most people don't develop these things until their late teens when the pre-frontal cortex is developed.
I believe the simple answer (vs complicated truth) is that the donor cell, a fertilized egg, is in a state accepting a not-quite-formed nucleus. There are no male cells that can get into that state AFAIK, possibly excepting pluripotent stem cells that are somehow convinced to undergo ovogenesis.
The truth is of course much more complicated than my limited understanding.
I encountered this in 1968. This was at Clarinda State School, in grade 1. The 1A class used ITA, and 1B used proper latin alphabet. My best friend, Steve Irwin (yes, _that_ Steve Irwin. Everyone had to go to school somewhere) was in 1A. Every afternoon after school he'd come to my place and we'd go through our readers for the day. I'd read the English one to him, and he'd memorise it to recite (pretending to read) to his teacher the next day. I assume he was taught properly when he moved to Queensland in 1969.
I can't imagine how hard it was for people less bright than Steve. No wonder the scheme trained illiterates.
Bright, but it sounds like he was also overcompensating. Many celebs, leaders in thier respective fields, get there by being massive good at something. Often they get that good as mental cover for some other self-percieved failure. The drive required to be the absolute best is itself rather unnatural, requiring some sort of trigger. Being a totally friendly extrovert seems a logical cover for poor reading ability.
I've taken my time to respond to this. Firstly, that's a big statement about someone you only know from TV.
Secondly, he was one of the most functional human beings I've ever met.
The reason he was so good at herpetology is because he was raised with dangerous reptiles. The backyard of his home in Botany Circuit was crawling with snakes, crocs and goannas. I rarely played at his place, because it un-nerved me. He was very confident and capable, and understood animal behaviour even at 6yo.
I was better at playing with spiders though, no doubt that changed as he grew older.
I've taken quite a lot of these. I was on 3600mg/day for about 5 years. I was later on lyrica for about 3 years.
These drugs _do_ help you to tolerate nerve pain, but do _not_ stop it. I found they led to my otherwise amazing memory developing "holes" - I knew I knew something, but I couldn't put my finger on it. As someone accused of having an eidetic memory (I'm not convinced) I found this really disturbing. I also felt more stupid, and less quick.
I've been off gabapentinoid medications for about 4 years. My pain is more "in my face", but I no longer feel retarded.
Note: The advice on the gabapentinoid box "May increase the effects of alcohol" is both warning and recommendation. It's not unknown for the mix to include open-eyed hallucinations.
It’s the top end of what gets prescribed for spinal nerve pain but not an uncommon dose. I was on the same. I spent the last year tapering off of it.
I’ll note I did not have the same experience. It eliminated my nerve pain and I did feel mentally sluggish when I first started but that receded and I didn’t have memory issues while on it.
In my experience as a medically complicated person, generics generally work, but may have different side effects, both in scale and style. One of the worst was a duloxetine generic that came on so hard and strong that I felt serontoninised.
My friend the John the Pharmacist explained that the binders etc can accelerate absorption. His advice was be careful the first two days of a new generic formulation.
I would assume the NHS (like the TGA here in Oz) looks _very_ carefully at the side-affect profile before they buy any particular generic. Government agencies tend to try not to poison voters.
I take dimethyl-fumarate, which is a serious medication for MS, but sadly ever since "Tecfidera" got withdrawn for some reason, I can only get a "shittier" generic. That said, side-effect profile appears to be the same, thankfully. All in all, I think they are "almost" identical, if not identical.
I’ve had decent luck with slow release generics, but they go out of their way to make SR generics take a quarter of a human lifetime to come out after the original is available.
The paragraph on the relation to Brugada Syndrome hits home, as I have that rare disease. What isn't clear is if it's safe for Brugada patients - a hell of a lot of drugs are not advisable for us. As I also have CRPS type II (complex regional pain syndrome) I'd like a non-narcotic strong pain killer that doesn't induce arrhythmia.
"For example, the Lotka-Volterra model accurately captures predator-prey dynamics using systems of differential equations."
This is incorrect. The validation of the L-V predator/prey model was considered to be the population dynamics of the Snow Shoe Hare and Canada Lynx as seen in Hudson Bay Company records. The data actually models the fashion cycles in Europe, showing prices and demand from Europe drove the efforts of the Company and the trappers. This is in the standard texts from at least the mid 90s AFAIK.
